hsa-miR-93-5p
Submitted by Anonymous on Wed, 10/30/2019 - 22:16
Pubmed
Gene symble | Title | PMID | Curated descirption | Category | Literature category |
---|---|---|---|---|---|
hsa-miR-93-5p | miR-93 inhibits the invasive potential of triple-negative breast cancer cells in vitro via protein kinase WNK1 | 27840899 | Further in vivo studies are required to ascertain the miR-93-WNK1-metastasis cascade, that has potential implications in breast cancer therapy | mechanism | BC/OC research |
hsa-miR-93-5p | MiR-93-5p inhibits the EMT of breast cancer cells via targeting MKL-1 and STAT3 | 28499590 | MiR-93-5p regulates MKL-1 and STAT3 to affect EMT controlling breast cancer cell migration | mechanism | BC/OC research |
hsa-miR-93-5p | MicroRNA-93 promotes proliferation and metastasis of gastric cancer via targeting TIMP2 | 29220395 | MiR-93 might be a promising biomarker and therapeutic target for treatment of gastric cancer | biomarker | BC/OC research |
hsa-miR-93-5p | MicroRNA-93 promotes proliferation and metastasis of gastric cancer via targeting TIMP2 | 29220395 | MiR-93 serves as a tumor promoter in human gastric carcinogenesis by targeting TIMP2 | mechanism | BC/OC research |
hsa-miR-93-5p | miR-93-5p Transferred by Exosomes Promotes the Proliferation of Esophageal Cancer Cells?via?Intercellular Communication by Targeting PTEN | 29673440 | Upregulation of plasma miR-93-5p expression significantly increases the risk of esophageal cancer and is associated with poor prognosis | biomarker | BC/OC research |
hsa-miR-93-5p | miR-93-5p Transferred by Exosomes Promotes the Proliferation of Esophageal Cancer Cells?via?Intercellular Communication by Targeting PTEN | 29673440 | MiR-93-5p transferred by exosomes promotes the proliferation of recipient esophageal cancer cells and affects the expression of PTEN and its downstream proteins p21 and cyclin D1 | mechanism | BC/OC research |
hsa-miR-93-5p | miR-93-5p enhance lacrimal gland adenoid cystic carcinoma cell tumorigenesis by targeting BRMS1L. | 29760585 | MiR-93 promotes LACC cell migration, invasion, and proliferation via targeting downregulation of BRMS1L through regulation of Wnt signaling pathway | mechanism | BC/OC research |
hsa-miR-93-5p | Differential expression of exosomal miRNAs between breast cancer patients with and without recurrence | 29050253 | Of 384 miRNAs, three miRNAs (miR-338-3p, miR-340-5p, and miR-124-3p) were significantly upregulated and eight (miR-29b-3p, miR-20b-5p, miR-17-5p, miR-130a-3p, miR-18a-5p, miR-195-5p, miR-486-5p, and miR-93-5p) were significantly downregulated in the patients with recurrence | biomarker | BCEXO research |
Survival analysis
Pathways
Enriched Terms | Cross Refer. |
---|---|
Protein processing in endoplasmic reticulum | KEGG |
Choline metabolism in cancer | KEGG |
Insulin signaling pathway | KEGG |
Focal adhesion | KEGG |
Thyroid hormone signaling pathway | KEGG |
Endometrial cancer | KEGG |
Non-small cell lung cancer | KEGG |
RNA degradation | KEGG |
Sphingolipid signaling pathway | KEGG |
PI3K-Akt signaling pathway | KEGG |