hsa-miR-3188
Submitted by Anonymous on Wed, 10/30/2019 - 22:16
Pubmed
Gene symble | Title | PMID | Curated descirption | Category | Literature category |
---|---|---|---|---|---|
hsa-miR-3188 | miR-3188 regulates nasopharyngeal carcinoma proliferation and chemosensitivity through a FOXO1-modulated positive feedback loop with mTOR–p-PI3K/AKT-c-JUN | 27095304 | As a tumour suppressor, miR-3188 directly targets mTOR to stimulate its own expression and participates in FOXO1-mediated repression of cell growth, tumorigenesis and NPC chemotherapy resistance | mechanism | BC/OC research |
hsa-miR-3188 | miR-3188 Regulates Cell Proliferation, Apoptosis, and Migration in Breast Cancer by Targeting TUSC5 and Regulating the p38 MAPK Signaling Pathway | 28560951 | MiR-3188 may serve as a potential therapeutic agent for the treatment of breast cancer | biomarker | BC/OC research |
hsa-miR-3188 | miR-3188 Regulates Cell Proliferation, Apoptosis, and Migration in Breast Cancer by Targeting TUSC5 and Regulating the p38 MAPK Signaling Pathway | 28560951 | MiR-3188 affects breast cancer cell proliferation, apoptosis, and migration by targeting TUSC5 and activating the p38 MAPK signaling pathway | mechanism | BC/OC research |
hsa-miR-3188 | Hepatitis B virus X protein promotes CREB-mediated activation of miR-3188 and Notch signaling in hepatocellular carcinoma | 28574502 | The HBx-miR-3188-ZHX2-Notch1 signaling pathway plays an important role in the pathogenesis and progression of HBV-related HCC with family history of HCC | mechanism | BC/OC research |
hsa-miR-3188 | MiR-3188 Inhibits Non-small Cell Lung Cancer Cell Proliferation Through FOXO1-Mediated mTOR-p-PI3K/AKT-c-JUN Signaling Pathway | 30618730 | MiR-3188 interacts with mTOR and FOXO1 to inhibit NSCLC cell proliferation through a mTOR-p-PI3K/AKT-c-JUN signaling pathway | mechanism | BC/OC research |
hsa-miR-3188 | Loss of exosomal miR-3188 in cancer-associated fibroblasts contributes to HNC progression | 30961650 | The potential therapeutic value of exosomal miR-3188 for inhibiting HNC growth. | mechanism | BC/OC research |
Survival analysis

Pathways
Enriched Terms | Cross Refer. |
---|---|
Ribosome | KEGG |